The apricot rickets disease is a typical physiological disease. There are many causes of the disease, such as bruises, insects, frostbite, sunburn, mechanical trauma, etc. , Summer pruning is too heavy, improper fertilization, soil viscous, acidic too strong, improper use of pesticides and caused by phytotoxicity can induce gum disease. Only by mastering the pathogenesis of the disease can we effectively prevent and treat it. Now we will introduce the symptoms, morbidity, and control techniques of the disease as follows: Symptoms of fluids occur mainly on branches and fruits. When the trunks and branches are damaged, spring outflow occurs. Transparent gum, after contact with the air, the gum gradually becomes brown, becomes a crystal soft plastic block, and finally becomes a dark brown hard plastic block. Flowing gum is often swollen and the cortex and xylem of the diseased part gradually become brown and rot, and is then infected by saprophytic bacteria, which severely weakens the tree vigor. The flow of fruit occurs mostly in the wound, and the glue sticks on the surface of the fruit, causing the growth of the fruit to stagnate and the quality to decline. Pathogens of pathogenic apricot tree flow gum disease are several species of Verticillium spp., which belong to the class of Deuteromycetes, Sphaerotheca fuliginea, and Stemella spp. It was initially determined that the pathogenic bacteria were saprophytic bacteria that had invaded after being flowed by physiological lesions, and that the invasion of the pathogens further aggravated the flow of glue. Pathogenesis The conidia of the pathogenic bacteria of the disease, through the spread of wind and rain, invade the wound or fluid. The pathogenic bacteria can lurk in the tissues of the victim's branches and xylem, producing conidia in the dead cortex and becoming a source of infection. Prevention methods (1) Avoid causing mechanical damage to the tree body. In the event of injury, promptly apply a lead oil preservative to the wound. (2) Eradicate pests in time and control the amount of nitrogen fertilizer. (3) In the dormant period, smear lesions with colloidal bactericide (formulated by 1 kg latex + 100 g 50% tetromycin) to kill pathogens. (Source: Zhao Li, Northern Vegetable Newspaper)
Piston Crown
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AAA) YANMAR DIESEL ENGINE
K(D)L, KFL-T, 6MAL-H/T/HT/DT, S165-T/ST/EN,
S185-ST/UT/ET, M200L-UN/SN/EN/M220L-UN/SN/EN / N 330
BBB) DAIHATSU DIESEL ENGINE
PS22/26/ DS18(A)/ DS(B)22/
DL20/22/28/ DK20 / DK 28
CCC) NIIGATA DIESEL ENGINE
6L(M)25BX/6M28AFTE/BX/6M31X/EZ/EX/ 31AFTE /34AGT
DDD) AKASAKA
AH30/AH38/ AH40/D/F/ A31/34/37/41/ DM28/30/33/36/38/40(A)K/ DM46/ UEC37H-HB/ UEC 37/88H
UEC 37LA/ UEC45LA/ UEC52LA/ UEC52HA/ UEC60HA/ UEC60LS/ UEC45HA/115 ALL UET TYPE
EEE) MITSUBISHI (KOBE AND AKASAKA )
45/75C/ 45/80D/ 52/90D/ 52/105D.E/
UEC37H-IIB/ UET37/88HA/UEC45HA.LA/ UEC52HA.LA/ S6B/
ETC
FFF) HANSHIN
6LU(D)26/ 6LU(N)28A/ 6LU(D)32/6LU(D)35/ 6LU(S)38/ 40 / 6LU40/ 6LU46A/ 6LU50A/
6EL30/ EL(S)32 /6EL(S)35/ 6EL38/ 6EL40/ 6EL(S)44/50A/
GGG) MAN B&W
S35MC/L35MCE/L50MCE/ L60MCE/ 40/54A
HHH) MITSUBISHI PURIFIER
SJ700~SJ1800/SJ 2000
III)SULZER
RND 68 RND 76 RD 44
JJJ) MAKITA
GSLH – 633 637 KSLH -633 637
Kkk) PIELSTICK
PC 2-5 PC 2-6
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